Excessive Alcohol Intake

Also, what is the highest penalties, you can get if the capture of a Claass a drug.
Science homework: (
Thank you!

I'm not rocket scientist, but basically if you drink large amounts of alcohol at one time, it can cause serious damage to your liver because it simply can not handle both, that's why people say, you should have serious obstacles between drinks. But neither drink alcohol to damage your liver in the long term, especially if you drink every day.

If you drink cirrhosis may occur http://digestive.niddk.nih.gov/ddiseases ... which is among many other diseases you might get from drinking

If you want to know more here is a useful PDF on the effects of alcohol on the live http://pubs.niaaa.nih.gov/publications/a ...

With the class A drug, I suppose if your caught with one, you would probably get one or all of the following: a $ 100-300 fine, being arrested, having to appear in court and have put in your criminal record. You might not even be able to travel to other countries, in some cases

Hope this helps your homework lol

a. changes in fatty liver
b. Alcoholic hepatitis
C. Cirrhosis

fatty change, or steatosis is the accumulation of fat in liver cells can be considered as fat globules under the microscope. Alcoholism causes large fat globules (macrovesicular steatosis). small fat globules have different causes. Other causes of macrovesicular steatosis include diabetes, obesity and starvation. alcoholic fatty change is probably related to dose. [Edit] The mechanism of alcohol induced fatty liver implies an above average NADH: NAD ratio caused by alcohol metabolism by alcohol dehydrogenase and aldehyde dehydrogenase. A higher concentration of NADH induces the synthesis of fatty acids decreased while that NAD levels decrease causes the oxidation of fatty acids. Therefore, higher levels of fatty acid compounds hepatocytes signals it with glycerol to form triglycerides.

Some people have an acute hepatitis or inflammatory reaction in the cells affected by fatty change. This is not directly related to the dose of alcohol. Some people seem more prone to this reaction than others. This is called alcoholic fat necrosis and inflammation probably predisposes liver fibrosis.

Cirrhosis is a late stage of liver disease characterized by fibrosis and hepatic architecture. It is often progressive and may eventually lead to liver failure. Late complications of cirrhosis or liver failure include portal hypertension, bleeding disorders, ascites and other complications, including hepatic encephalopathy and hepatorenal syndrome.

Cirrhosis also causes many more abuse of alcohol, such as viral hepatitis and non-alcoholic toxins. The last stages of cirrhosis may resemble whatever the cause. This phenomenon is called "final common pathway" of the disease.

fatty change and alcoholic hepatitis are probably reversible. Later stages of fibrosis and cirrhosis tend to be irreversible, but can generally be well managed for long periods of time.

As the scars of the liver, blood vessels become narrow and non-compliant. This leads to increased pressure in blood vessels enter the liver. Over time, this causes a back log of blood (portal hypertension) and is associated with massive hemorrhage. Enlarged veins also develop to bypass blockages in the liver. These veins are very fragile and tend to rupture and bleed (varicose veins). variceal bleeding can be life threatening and needs emergent treatment. Once the liver is damaged, fluid accumulates in the abdomen and legs. The f.