Drinking And Breast Cancer

Breast cancer occurs when cells divide breast and grow uncontrolled. The cell cycle is the natural mechanism that regulates growth and cell death. When normal cells and cells do not die regulators dysfunction in the appropriate dose, uncontrolled cell growth and cancer will develop. breast cancer tumors usually grow slowly. When a tumor is large enough to be felt as a lump, it may have been more than 10 years and the spread of tumor cells (metastasis) may have occurred. Therefore, the screening methods (mammography through, ultrasound, MRI, or thermography) are very important. In addition, preventive measures such as healthy eating and living, food supplements and exercise are essential.

What are the risk factors for breast cancer?

It is useful to classify the modifiable risk factors for those who can not be changed. Most risk factors for breast cancer are modifiable, meaning that they can be modified according to the daily choices regarding nutrition, exercise, lifestyle and stress management.

Non-modifiable risk factors:

• Female
• Advancing age
• Family History (BRCA1 or 2)
• early age at menarche (first menstrual cycle)
• late menopause
• Diethylstilbestrol (DES) use by the mother

modifiable risk factors:

• Obesity (nearly triple the risk)
• Lack of exercise
• Synthetic HRT, particularly progestins
• the use of birth pill
• animals and high trans fat / low fiber / low fruit and vegetable diet
• Smoking
• Breast trauma
• pregnancy advanced age, never having been pregnant, lack of breastfeeding
• Heavy alcohol consumption (> 1 glass per day)
• Cigarettes
• Working the graveyard "shift environmental toxins (radiation, xenoestrogens, secondhand smoke)
• Benign breast disease (fibrocystic breast changes or disease may or may not increase the risk)

Can you explain the effects of various hormones on breast tissue?

Estrogens, especially estradiol and estrone, stimulate proliferation of breast tissue and breast cancer cells. There is much confusion about the use of estrogen replacement therapy (ERT) and risk of breast cancer, which is true for the use of human identical (bio-identical) estrogens and the synthetic forms, such as Premarin. It is useful to examine the results of 3 large, well-conducted studies in recent years - the Women's Health Initiative, the Million Women Study and the Nurses' Health Study - to clarify the relationship between estrogen replacement and the risk of breast cancer. The Women's Health Initiative study (JAMA 2002) found an increased risk of breast cancer among women using Premarin with Provera, synthetic progestins, but not in women using Premarin alone. The Million Women Study (Lancet 2003) showed an increased risk with ERT (in all its forms, except intravaginal estriol) and an even higher risk with estrogen synthetic progestins.Lastly addition, the Nurses' Health Study (NEJM 1995) found similar results, higher risk of breast cancer with ERT, and much higher when synthetic progestins have been added.

It is important to note that several studies have found no increased risk with the use of bio-identical estriol, particularly when used vaginally (Bergkvist 1989, Million Women Study 2003, Fournier 2004, Rosenberg 2006, Lyytinen 2006). In addition, use vaginal estriol in survivors of breast cancer does not increase the risk of recurrence or death (Dew 2003)

The data are compelling that synthetic progestins (eg Provera) significantly increased the risk of breast cancer. However, the same man (bioidentical) progesterone is a molecule different from that of synthetic progestins and a.